This new study finds that Bee Venom Therapy (BVT) has positive results on ALS symptoms, but those who've been using BVT for ALS, MS and various forms of Arthritis can already attest to its benefits. This is promising news for ALS sufferers, as use of the other bee products have been proven to aid and prevent neurodegenerative diseases.
To investigate
the effects of melittin on inflammation in the lungs and spleen, we used
hSOD1(G93A) transgenic mice that are mimic for ALS. Melittin treatment reduced
the expression of inflammatory proteins, including Iba-1 and CD14 by 1.9- and
1.3-fold (p < 0.05), respectively, in the lungs of symptomatic hSOD1(G93A)
transgenic mice. In the spleen, the expression of CD14 and COX2 that are
related to inflammation were decreased by 1.4 fold (p < 0.05) and cell
survival proteins such as pERK and Bcl2 were increased by 1.3- and 1.5-fold (p
< 0.05) in the melittin-treated hSOD1G93A transgenic mice.
Melittin
ameliorates the inflammation of organs in an amyotrophic lateral sclerosis
animal model
J Neuroinflammation,
2011 June
Amyotrophic
lateral sclerosis (ALS) is a devastating progressive neurodegenerative disorder
characterized by a selective loss of motor neurons in the spinal cord,
brainstem, and motor cortex, leading to weakness of the limb and bulbar
muscles. Although the immediate cause of death in ALS is the destruction of
motor neurons, ALS is a multi-organ disease that also affects the lungs,
spleen, and liver. Melittin is one of components of bee venom and has
anti-neuroinflammatory effects in the spinal cord, as shown in an ALS animal
model.
To investigate
the effects of melittin on inflammation in the lungs and spleen, we used
hSOD1(G93A) transgenic mice that are mimic for ALS. Melittin treatment reduced
the expression of inflammatory proteins, including Iba-1 and CD14 by 1.9- and
1.3-fold (p < 0.05), respectively, in the lungs of symptomatic hSOD1(G93A)
transgenic mice. In the spleen, the expression of CD14 and COX2 that are
related to inflammation were decreased by 1.4 fold (p < 0.05) and cell
survival proteins such as pERK and Bcl2 were increased by 1.3- and 1.5-fold (p
< 0.05) in the melittin-treated hSOD1G93A transgenic mice.
These findings
suggest that melittin could be a candidate to regulate the immune system in
organs affected by ALS.
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